• Open Daily: 10am - 10pm
    Alley-side Pickup: 10am - 7pm

    3038 Hennepin Ave Minneapolis, MN
    612-822-4611

Open Daily: 10am - 10pm | Alley-side Pickup: 10am - 7pm
3038 Hennepin Ave Minneapolis, MN
612-822-4611
Shock Pathogenesis and Therapy: An International Symposium. Stockholm, June 27th-30th 1961 Sponsored by CIBA

Shock Pathogenesis and Therapy: An International Symposium. Stockholm, June 27th-30th 1961 Sponsored by CIBA

Paperback

Medical Reference

ISBN10: 3662224054
ISBN13: 9783662224052
Publisher: Springer
Published: Nov 19 2013
Pages: 409
Weight: 1.07
Height: 0.86 Width: 5.50 Depth: 8.50
Language: English
1. Renal failure following circulatory shock develops because of per- sistent vasoconstriction which is just sufficient to prevent glomerular filtration. Hypoxia of renal tissue has not been demonstrated in surviving cases. 2. During the low-pressure phase in circulatory shock the remaining blood flow through the medullary regions washes out the osmotic gradient built up by the countercurrent system of Henle's loops, so that a concentrated urine cannot be formed. 3. During recovery from circulatory failure the osmotic gradient of the medullary region can only be built up if sufficient fluid from the glomerular filtrate reaches the countercurrent system. The greater the GF, the faster the gradient is built up. References 1. BoYLAN, J. W., and E. AssHAUER: Unpublished data. -2. DEETJEN, P., and K. KRAMER: Pfliigers Arch. Physiol. (G. ) (in press). - 3. KRAMER, K., and K. ULLRICH: Pfliigers Arch. Physiol. (G. ) 267,251 (1958). -4. KRA- MER, K., K. THURAU and P. DEETJEN: Pfliigers Arch. Physiol. (G. ) 270, 251 (1960). - 5. KRAMER, K., and P. DEETJEN: Pfliigers Arch. Physiol. (G. ) 271, 782 (1960). -6. KRAMER, K., and P. DEETJEN: Unpublished data. - 7. KuHN, W., and A. RAMEL: Helv. chim. acta 42,628 (1959). - 8. LASSEN, N. A., 0. MuNcK and J. H. THAYSEN: Acta physiol. Scand. IJ1, 371 (1961). - 9. MUNCK, 0.: Renal Circulation in Acute Renal Failure. Oxford 1958.

Also in

Medical Reference