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Mutationen in der mt-DNA von Spermien irakischer Asthenozoospermie-Patienten
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ISBN10: 6207549414
ISBN13: 9786207549412
Publisher: Verlag Unser Wissen
Published: May 16 2024
Pages: 156
Weight: 0.52
Height: 0.36 Width: 6.00 Depth: 9.00
Language: German
ISBN13: 9786207549412
Publisher: Verlag Unser Wissen
Published: May 16 2024
Pages: 156
Weight: 0.52
Height: 0.36 Width: 6.00 Depth: 9.00
Language: German
Die Mutationsrate der mitochondrialen DNA ist 10-20 Mal höher als die der Kern-DNA, was auf die ungeschlechtliche Replikationsmethode, einen sehr einfachen Reparaturmechanismus, das Fehlen von schützenden Histonen und die grö ere Nähe zum Ort der Entstehung freier Radikale zurückzuführen ist. Im Mittelstück von Säugetierspermien befinden sich 70-80 Mitochondrien. Es ist seit langem bekannt, dass sich in jedem Mitochondrium im Mittelstück von Säugetierspermien eine Kopie der mtDNA befindet. Asthenozoospermie ist definiert als eine Spermienmotilität von weniger als 50-60 % oder eine Abnahme der linearen progressiven Bewegungsmerkmale. Der Gehalt an mitochondrialer DNA kann als nützlicher Indikator für die Spermienqualität dienen, und die Verarmung der mt-DNA könnte eine wichtige Rolle in der Pathophysiologie mancher männlicher Unfruchtbarkeit spielen. Die Mutationen in der mt-DNA spielen eine entscheidende Rolle bei der Unfruchtbarkeit. Künftige Studien müssen sich auf die Hauptursachen der mt-DNA für männliche Unfruchtbarkeit konzentrieren, die Mutationen und Deletionen verursachen, um diese Gründe zu vermeiden.
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